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As these words suggest, Takeda's pharmaceutical business is involved in health and life for all. The Company is engaged in various fields of medicine and contributes to society by continuously creating quality pharmaceuticals for the benefit of patients and healthcare professionals. From an even longer-term perspective, we have defined the following five basic goals as our management vision. We aim to become: 1 ; A multinational company, driven by research and development, which leads the world through its unique strengths. 2 ; A company with highly integrated global operations. 3 ; A company that meets the needs of people around the world through superior products and services. 4 ; A company that grows together with its shareholders and other stakeholders. 5 ; An energetic company that attracts and retains wellqualified personnel from all over the world. The management mission and vision set the direction for the entire Takeda Group. They promote group-wide solidarity, and serve as a special message to Takeda stakeholders worldwide. In the future, we will develop optimal business strategies with a long-term and global focus, based on this mission and vision. We will build operating structures that enable the speedy, efficient, and systematic implementation of these strategies. Realizing these objectives will be difficult without the support and encouragement of you, our shareholders. At the Board of Directors meeting held on May 17, 2002, changes were approved to our minimum stock trading unit that I expect will facilitate greater investor participation in Takeda's global development. The resolution was approved to lower the number of shares in one trading unit from 1, 000 shares to 100 shares, effective from September 2, 2002. By implementing the 20012005 Medium-term Management Plan, we will make steady progress in our reforms and strategies aimed at enhancing Takeda's position as a world-class pharmaceutical company. As well as ensuring our operations comply with regulations and ethics, we will work to realize our management mission and vision and maximize shareholder value. TABLE 2. Comparison of genotypic and phenotypic drug resistance results for respiratory and nonrespiratory specimens, because what is metrogel vaginal.
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2005-2006 SOMERVILLE HIGH SCHOOL HEALTH SURVEY Mental Health S High School N.1, 003. The Sara Koe PSP Research Centre was officially opened by HRH The Duchess of Gloucester on the 17th April 2002. The Duchess is shown observing tissue under the microscope. Also shown are members of the family of the late Sara Koe and Professor Richard Frackowiak on right ; . SPTLC1 gene in HSAN 1 Dr H. Houlden ; . Research into CMT has included the identification of new mutations in the connexin 32 gene Drs M. Lee and W. Leong ; and the identification of a novel mutation in the promoter of the connexin 32 gene Dr H. Houlden ; . In collaboration with Professor M. Shy Department of Neurology, Wayne State University, Detroit, USA ; and Dr N. Murray Department of Neurophysiology, NHNN ; , we have studied nerve conduction velocities in CMT 1A and developed a clinical scale for CMT patients. Studies into rare genetic neuropathies have included the identification of mutations in the genes for giant axonal neuropathy and Triple A syndrome Dr H. Houlden ; . Dr W. Leong is completing a large clinical, neurophysiological, pathological and genetic study of nearly 100 patients with CIDP and multifocal motor neuropathy with conduction block MMN ; patients and Dr M. Laura has undertaken MRI scans of brain and spinal cord in collaboration with Professor D. Miller ; , on a sub-group of these patients and shown some interesting findings in the cervical cord in CIDP patients. The Group has had a long-term interest in the role of mitochondrial dysfunction in muscle disease and this work has been enhanced by the funding through a component grant of the MRC cooperative "Mitochondria in Health and Disease". We have been able to define a new disease causing mutations in human mtDNA and we have obtained evidence that adult mitochondrial disease may also be caused by nuclear gene mutations. We have also developed cell and microcell mediated chromosome strategies to identify such nuclear genes. These research activities into neuromuscular disease will complement the development of a centre for neuromuscular disease, which is being established by the NHNN. Molecular pathogenesis of paroxysmal disorders of the nervous system and mobic.
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